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Category Names, Results, and Descriptions

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GABA Agents (79) • Substances used for their pharmacological actions on GABAergic systems. GABAergic agents include agonists, antagonists, degradation or uptake inhibitors, depleters, precursors, and modulators of receptor function. MeSH

GABA Agonists (17) • Endogenous compounds and drugs that bind to and activate GAMMA-AMINOBUTYRIC ACID receptors (RECEPTORS, GABA). MeSH

GABA Antagonists (19) • Drugs that bind to but do not activate GABA RECEPTORS, thereby blocking the actions of endogenous GAMMA-AMINOBUTYRIC ACID and GABA RECEPTOR AGONISTS. MeSH

GABA Effect (0) see GABA Agents

GABA Modulators (37) • Substances that do not act as agonists or antagonists but do affect the GAMMA-AMINOBUTYRIC ACID receptor-ionophore complex. GABA-A receptors (RECEPTORS, GABA-A) appear to have at least three allosteric sites at which modulators act: a site at which BENZODIAZEPINES act by increasing the opening frequency of GAMMA-AMINOBUTYRIC ACID-activated chloride channels; a site at which BARBITURATES act to prolong the duration of channel opening; and a site at which some steroids may act. GENERAL ANESTHETICS probably act at least partly by potentiating GABAergic responses, but they are not included here. MeSH

GABA-A Receptor Agonists (5) • Endogenous compounds and drugs that bind to and activate GABA-A RECEPTORS. MeSH

GABA-A Receptor Antagonists (2) • Drugs that bind to but do not activate GABA-A RECEPTORS thereby blocking the actions of endogenous or exogenous GABA-A RECEPTOR AGONISTS. MeSH

GABA-B Receptor Agonists (2) • Endogenous compounds and drugs that bind to and activate GABA-B RECEPTORS. MeSH

GABA-B Receptor Antagonists (1) • Drugs that bind to but do not activate GABA-B RECEPTORS thereby blocking the actions of endogenous or exogenous GABA-B RECEPTOR AGONISTS. MeSH

Ganglionic Blockers (9) • Agents having as their major action the interruption of neural transmission at nicotinic receptors on postganglionic autonomic neurons. Because their actions are so broad, including blocking of sympathetic and parasympathetic systems, their therapeutic use has been largely supplanted by more specific drugs. They may still be used in the control of blood pressure in patients with acute dissecting aortic aneurysm and for the induction of hypotension in surgery. MeSH

Ganglionic Stimulants (5) • Agents that mimic neural transmission by stimulation of the nicotinic receptors on postganglionic autonomic neurons. Drugs that indirectly augment ganglionic transmission by increasing the release or slowing the breakdown of acetylcholine or by non-nicotinic effects on postganglionic neurons are not included here nor are the nonspecific cholinergic agonists. MeSH

Gasotransmitters (3) • Endogenously produced lipid-soluble gaseous molecules which function as neurotransmitters and signal mediators targeting ION CHANNELS and transporters. MeSH

Gastric Agents (0) see Gastrointestinal Agents

Gastrointestinal Agents (254) • Drugs used for their effects on the gastrointestinal system, as to control gastric acidity, regulate gastrointestinal motility and water flow, and improve digestion. MeSH

Gelatinase Inhibitors (0) see Matrix Metalloproteinase Inhibitors

Gels (1)

Glucocorticoid Effect (0) see Glucocorticoids

Glucocorticoid Synthesis Inhibitors (0) see Steroid Synthesis Inhibitors

Glucocorticoids (52) • A group of CORTICOSTEROIDS that affect carbohydrate metabolism (GLUCONEOGENESIS, liver glycogen deposition, elevation of BLOOD SUGAR), inhibit ADRENOCORTICOTROPIC HORMONE secretion, and possess pronounced anti-inflammatory activity. They also play a role in fat and protein metabolism, maintenance of arterial blood pressure, alteration of the connective tissue response to injury, reduction in the number of circulating lymphocytes, and functioning of the central nervous system. MeSH

Glutamate Agonists (0) see Excitatory Amino Acid Agonists

Glycine Agents (2) • Substances used for their pharmacological actions on glycinergic systems. Glycinergic agents include agonists, antagonists, degradation or uptake inhibitors, depleters, precursors, and modulators of receptor function. MeSH

Glycine Effect (0) see Glycine Agents

Glycoside Hydrolase Inhibitors (13) • Compounds that inhibit or block the activity of GLYCOSIDE HYDROLASES such as ALPHA-AMYLASES and APHA-GLUCOSIDASES. MeSH

Goitrogens (0) see Antithyroid Agents

Gout Suppressants (22) • Agents that increase uric acid excretion by the kidney (URICOSURIC AGENTS), decrease uric acid production (antihyperuricemics), or alleviate the pain and inflammation of acute attacks of gout. MeSH

Growth Inhibitors (32) • Endogenous or exogenous substances which inhibit the normal growth of human and animal cells or micro-organisms, as distinguished from those affecting plant growth (= PLANT GROWTH REGULATORS). MeSH

Growth Substances (148)

GTP Phosphohydrolase Activators (0) • Agents and factors that activate GTP phosphohydrolase activity. MeSH

 
 
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